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Prevention of expira-tion by airway occlusion maintains vagal afferentinput, thus prolonging expiratory time.

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Using the most basic def-inition, three stages, initiation, promotion, and progression,have been shown (Figure 15.4). They are lined with transitionalepithelium (urothelium), an impervious layer that lines the urinaryexcretory passages from the renal calyces through the urethra.

Gap junctions between cells areresponsible for the transfer of small (less than 1kD) molecules such as small metabolites,ions, IP3, prostaglandins, ATP, and cAMP. Other described causes of Type II LPR include congenital and acquired mito-chondrial dysfunction, sepsis, citric acid cycle enzymatic abnormalities, hyperammonemia,seizures, increased glycogenolysis from medication-induced metabolism, and halothaneand other anesthetic/hypnotic use

Other described causes of Type II LPR include congenital and acquired mito-chondrial dysfunction, sepsis, citric acid cycle enzymatic abnormalities, hyperammonemia,seizures, increased glycogenolysis from medication-induced metabolism, and halothaneand other anesthetic/hypnotic use.

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